Author

Mary J. Feigman

Cold Spring Harbor Laboratory

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Developmental Biology· 1 Molecular Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Research Focus:gene regulatory network inference· 2 Organism:mouse· 2 Approach:statistical model· 2 Research Focus:cell-cell communication· 1 Assay:ChIP-seq· 1 Assay:DNA methylation· 1

Frequent Co-Authors

Camila O. dos SantosiD· 2 shared Chen CheniD· 2 shared John Erby WilkinsoniD· 2 shared Matthew A. MossiD· 2 shared Michael Francis Ciccone· 2 shared Samantha L. CyrilliD· 2 shared Amritha Varshini Hanasoge SomasundaraiD· 1 shared Jude Kendall· 1 shared

Activity by Year

2019-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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2 shared papersDevelopmental Biology

Camila O. dos SantosORCID

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2 papers

Recent·Most cited

Parity-induced changes to mammary epithelial cells control NKT cell expansion and mammary oncogenesis

Amritha Varshini Hanasoge Somasundara, Matthew A. Moss, Mary J. Feigman, Chen Chen, Samantha L. Cyrill, Michael Francis Ciccone, Marygrace C. Trousdell, Macy Vollbrecht, Siran Li, Jude Kendall, Semir Beyaz, John Erby Wilkinson, Camila O. dos Santos

The paper shows pregnancy-induced changes in mammary epithelial cells lead to NKT cell expansion and promote mammary oncogenesis through CD1d expression.

Cell Reports · Dec 1, 2021Peer-reviewedDevelopmental Biology

Pregnancy reprograms the enhancer landscape of mammary epithelial cells and alters the response to cMYC-driven oncogenesis

Mary J. Feigman, Matthew A. Moss, Chen Chen, Samantha L. Cyrill, Michael Francis Ciccone, Wesley D. Frey, Shih Ting Yang, John Erby Wilkinson, Camila O. dos Santos

The paper investigates how pregnancy alters the enhancer landscape of mammary epithelial cells and affects their response to cMYC-driven oncogenesis, finding that post-pregnancy cells are resistant to cMYC-induced proliferation.

bioRxiv · May 20, 2019 · PreprintMolecular Biology