Author

Minza Haque

Research Institute of the McGill University Health Centre

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Neuroscience· 1 Synthetic Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Assay:CRISPR screen· 1 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

Wei‐Hsiang HuangiD· 2 shared Yu‐Ju LeeiD· 2 shared Connie AniD· 1 shared Hao‐Cheng Chang· 1 shared Jeewoo KangiD· 1 shared Josh TyckoiD· 1 shared Lacramioara BintuiD· 1 shared Meng ZhangiD· 1 shared

Activity by Year

2023-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 38 mutual co-author links

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2 shared papersSynthetic Biology

Wei‐Hsiang HuangORCID

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2 papers

Recent·Most cited

Tunable, proteolytic dosage control of CRISPR-Cas systems enables precise gene therapy for dosage sensitive disorders

Noa Katz, Connie An, Minza Haque, Sophia Mahiout, Yu‐Ju Lee, Josh Tycko, Meng Zhang, Jeewoo Kang, Lacramioara Bintu, Michael Cory Bassik, Wei‐Hsiang Huang, Xiaojing J. Gao

This paper introduces an IFFL-based CRISPR-Cas system to reduce gene expression variability, demonstrating improved dosage control for RAI1 in a mouse model.

bioRxiv · Oct 9, 2024 · PreprintSynthetic Biology

Smith-Magenis syndrome protein RAI1 regulates body weight homeostasis through hypothalamic BDNF-producing neurons and neurotrophin downstream signalling

Sehrish Javed, Ya-Ting Chang, Yoobin Cho, Yu‐Ju Lee, Hao‐Cheng Chang, Minza Haque, Yu Cheng Lin, Wei‐Hsiang Huang

This study shows that RAI1 regulates body weight through hypothalamic BDNF neurons and neurotrophin signaling, and that targeting this pathway can delay obesity in SMS mice.

eLife · Aug 23, 2023Peer-reviewedNeuroscience