Author

Mo Singer

Barrow Neurological Institute

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Genomics· 1 Neuroscience· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 2 Organism:human· 2 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Assay:scRNA-seq· 1 Approach:statistical model· 1

Frequent Co-Authors

Amanda McQuadeiD· 1 shared Amber Logemann· 1 shared Benjamin E. RabichowiD· 1 shared Camelia BurciuiD· 1 shared Chiara ScopaiD· 1 shared Christopher J. DonnellyiD· 1 shared Davide TrottiiD· 1 shared Deepti Lall· 1 shared

Activity by Year

2020-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperNeuroscience

Amanda McQuadeORCID

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

JUN upregulation drives aberrant transposable element mobilization, associated innate immune response, and impaired neurogenesis in Alzheimer disease

Chiara Scopa, Samantha M. Barnada, M.E. Cicardi, Mo Singer, Davide Trotti, Marco Trizzino

This study identifies JUN upregulation as a driver of transposable element mobilization, innate immune activation, and impaired neurogenesis in Alzheimer's disease.

bioRxiv · Nov 24, 2022 · PreprintGenomics

Cellular and molecular phenotypes of C9orf72 ALS/FTD patient derived iPSC-microglia mono-cultures

Ileana Lorenzini, Eric B. Alsop, Jennifer Levy, Lauren M. Gittings, Deepti Lall, Benjamin E. Rabichow, Stephen R. Moore, Ryan S. Pevey, Lynette M. Bustos, Camelia Burciu, Divya Bhatia, Mo Singer, Justin Saul, Amanda McQuade, Makis Tzioras, , Rita Sattler

This study examines C9orf72 ALS/FTD patient-derived iPSC-microglia mono-cultures, finding they form RNA foci, have reduced C9orf72 protein, and show endosomal-lysosomal dysfunction, but no significant microglial-enriched gene expression differences.

bioRxiv · Sep 3, 2020 · PreprintNeuroscience