Author

N. Felipe-Medina

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Approach:biophysical modeling· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:development & morphogenesis· 1 Research Focus:host-pathogen & immune response· 1 Assay:proteomics· 1

Frequent Co-Authors

A. M. Pendas· 2 shared A. Iborra· 1 shared A. Pujol· 1 shared C. Buza· 1 shared C. Coves· 1 shared C. Gallego-Paramo· 1 shared C. Madrid-Sandin· 1 shared E. Llano· 1 shared

Activity by Year

2020-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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2 shared papersCell Biology

A. M. Pendas

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

The full length BEND2 protein is dispensable for spermatogenesis but required for setting the ovarian reserve in mice

Y. Huang, N. Bucevic, C. Coves, N. Felipe-Medina, M. Marcet-Ortega, N. Nikou, C. Madrid-Sandin, M. Lopez-Panades, C. Buza, N. Ferrer Miralles, A. Iborra, A. Pujol, A. M. Pendas, I. Roig

This study shows that full-length BEND2 is not needed for male fertility but is essential for establishing the ovarian reserve in mice.

bioRxiv · Feb 6, 2024 · PreprintCell Biology

Meiotic chromosome synapsis depends on multivalent SYCE1-SIX6OS1 interactions that are disrupted in cases of human infertility

F. Sanchez-Saez, L. Gomez-H, O. M. Dunne, C. Gallego-Paramo, N. Felipe-Medina, M. A. Sanchez-Martin, E. Llano, A. M. Pendas, O. R. Davies

This study identifies SYCE1-SIX6OS1 interactions critical for meiotic synapsis, showing that mutations disrupting these interactions cause human infertility.

bioRxiv · Feb 4, 2020Biochemistry