Author

M. A. Sanchez-Martin

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 1 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Organism:mouse· 2 Approach:biophysical modeling· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:drug response / drug discovery· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

A. M. Pendas· 1 shared C. Gallego-Paramo· 1 shared E. Llano· 1 shared E. Sebastian· 1 shared E. Vuelta· 1 shared F. Sanchez-Guijo· 1 shared F. Sanchez-Saez· 1 shared I. Garcia-Tunon· 1 shared

Activity by Year

2020-2020 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperBiochemistry

A. M. Pendas

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

CRISPR/Cas9 technology abolishes the BCR/ABL1 oncogene effect in chronic myeloid leukemia and restores normal hematopoiesis

E. Vuelta, J. L. Ordonez, V. Alonso-Perez, L. Mendez, P. Hernandez-Carabias, R. Saldana, J. Sevilla, E. Sebastian, S. Muntion, F. Sanchez-Guijo, J. M. Hernandez-Rivas, I. Garcia-Tunon, M. A. Sanchez-Martin

This study uses CRISPR/Cas9 to target and disrupt the BCR/ABL1 oncogene in CML cells, restoring normal hematopoiesis in mouse models and patient-derived xenografts.

bioRxiv · Aug 5, 2020 · PreprintCancer Biology

Meiotic chromosome synapsis depends on multivalent SYCE1-SIX6OS1 interactions that are disrupted in cases of human infertility

F. Sanchez-Saez, L. Gomez-H, O. M. Dunne, C. Gallego-Paramo, N. Felipe-Medina, M. A. Sanchez-Martin, E. Llano, A. M. Pendas, O. R. Davies

This study identifies SYCE1-SIX6OS1 interactions critical for meiotic synapsis, showing that mutations disrupting these interactions cause human infertility.

bioRxiv · Feb 4, 2020Biochemistry