Author

A. M. Pendas

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 3 Biochemistry· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Organism:mouse· 4 Research Focus:cell type annotation· 2 Research Focus:chromatin & epigenetic regulation· 2 Research Focus:development & morphogenesis· 2 Approach:biophysical modeling· 1 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1

Frequent Co-Authors

E. Llano· 3 shared L. Gomez-H· 2 shared N. Felipe-Medina· 2 shared A. D. Rolland· 1 shared A. Iborra· 1 shared A. Pujol· 1 shared A. Toth· 1 shared C. A. Eyster· 1 shared

Activity by Year

2019-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 18 mutual co-author links

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3 shared papersCell Biology

E. Llano

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Genome-wide transcriptional silencing and mRNA stabilization allow the coordinated expression of the meiotic program in mice

L. Bellutti, E. C. S. Peng, M.-J. Guerquin, S. Messiaen, E. Llano, V. Cluzet, I. Dereli, A. D. Rolland, E. Martini, A. Toth, A. M. Pendas, F. Chalmel, G. Livera

The paper quantifies transcriptional dynamics in mouse spermatogonia during meiosis, identifying a transcriptional slowdown and mRNA stabilization event that coordinates meiotic gene expression.

bioRxiv · Feb 8, 2024Cell Biology

The full length BEND2 protein is dispensable for spermatogenesis but required for setting the ovarian reserve in mice

Y. Huang, N. Bucevic, C. Coves, N. Felipe-Medina, M. Marcet-Ortega, N. Nikou, C. Madrid-Sandin, M. Lopez-Panades, C. Buza, N. Ferrer Miralles, A. Iborra, A. Pujol, A. M. Pendas, I. Roig

This study shows that full-length BEND2 is not needed for male fertility but is essential for establishing the ovarian reserve in mice.

bioRxiv · Feb 6, 2024 · PreprintCell Biology

Meiotic chromosome synapsis depends on multivalent SYCE1-SIX6OS1 interactions that are disrupted in cases of human infertility

F. Sanchez-Saez, L. Gomez-H, O. M. Dunne, C. Gallego-Paramo, N. Felipe-Medina, M. A. Sanchez-Martin, E. Llano, A. M. Pendas, O. R. Davies

This study identifies SYCE1-SIX6OS1 interactions critical for meiotic synapsis, showing that mutations disrupting these interactions cause human infertility.

bioRxiv · Feb 4, 2020Biochemistry

Shugoshin protects centromere pairing and promotes segregation of non-exchange partner chromosomes in meiosis

L. Previato, J. M. Evatt, H. H. Chuong, E. L. Kurdzo, C. A. Eyster, M. N. Gladstone, L. Gomez-H, E. Llano, R. Meyer, A. M. Pendas, R. J. Pezza, D. S. Dawson

This paper shows that shugoshin protects centromeric synaptonemal complex components and promotes proper segregation of non-crossover partner chromosomes in meiosis.

Proceedings of the National Academy of Sciences · Apr 24, 2019Peer-reviewedCell Biology