Author

N. Ronkina

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 1 Research Focus:development & morphogenesis· 1 Approach:experimental assay· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

A. Kotlyarov· 2 shared M. Gaestel· 2 shared A. Doerrie· 1 shared A. Gossler· 1 shared A. Krueger· 1 shared F. Boudghene-Stambouli· 1 shared F. Hansmann· 1 shared H. Kunze-Schumacher· 1 shared

Activity by Year

2018-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersCancer Biology

A. Kotlyarov

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2 papers

Recent·Most cited

On the therapeutic potential of MAPK4 in triple-negative breast cancer

F. Boudghene-Stambouli, M. Soulez, N. Ronkina, A. Doerrie, A. Kotlyarov, O.-M. Seternes, M. Gaestel, S. Meloche

The paper critiques Wang et al.'s study on ERK4's role in TNBC, arguing that methodological flaws undermine their conclusion that ERK4 is a therapeutic target.

bioRxiv · Aug 24, 2022 · PreprintCancer Biology

Germline deletion reveals a non-essential role of the atypical MAPK6/ERK3

N. Ronkina, K. Schuster-Gossler, F. Hansmann, H. Kunze-Schumacher, I. Sandrock, T. Yakovleva, J. Lafera, W. Baumgärtner, A. Krueger, I. Prinz, A. Gossler, A. Kotlyarov, M. Gaestel

The paper shows that ERK3 is not essential for viability, fertility, or T-cell function, contradicting previous reports of perinatal lethality due to a different genetic construct.

bioRxiv · Nov 10, 2018Cell Biology