Author

I. Sandrock

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Immunology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 3 Organism:mouse· 3 Assay:imaging· 2 Research Focus:cell type annotation· 1 Research Focus:development & morphogenesis· 1 Research Focus:drug response / drug discovery· 1 Assay:proteomics· 1

Frequent Co-Authors

I. Prinz· 2 shared A. Freudenstein· 1 shared A. Gossler· 1 shared A. J. M. Watson· 1 shared A. Kotlyarov· 1 shared A. Krueger· 1 shared A. Kupke· 1 shared A. Tscherne· 1 shared

Activity by Year

2018-2021 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 17 mutual co-author links

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Curved chords = mutual co-authorship

2 shared papersImmunology

I. Prinz

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

γδ intraepithelial lymphocytes facilitate pathological epithelial cell shedding via CD103-mediated granzyme release.

M. D. Hu, N. B. Golovchenko, T. J. Kelly, J. Agos, M. R. Zeglinski, E. M. Bonder, I. Sandrock, I. Prinz, D. J. Granville, A. J. M. Watson, K. L. Edelblum

This study identifies γδ intraepithelial lymphocytes as facilitators of pathological epithelial cell shedding through CD103-mediated granzyme release in response to LPS.

bioRxiv · Jan 20, 2021Immunology

Immunogenicity and efficacy of the COVID-19 candidate vector vaccine MVA SARS 2 S in preclinical vaccination

A. Tscherne, J. H. Schwarz, C. Rohde, A. Kupke, G. Kalodimou, L. Limpinsel, N. M. A. Okba, B. Bosnjak, I. Sandrock, S. Halwe, L. Sauerhering, K. Printz, L. Nan, E. Duell, S. Jany, , A. Volz

This paper describes the preclinical evaluation of MVA-SARS-2-S, a vector vaccine against SARS-CoV-2, showing it induces immune responses and protects mice from infection.

bioRxiv · Jan 9, 2021Microbiology

Germline deletion reveals a non-essential role of the atypical MAPK6/ERK3

N. Ronkina, K. Schuster-Gossler, F. Hansmann, H. Kunze-Schumacher, I. Sandrock, T. Yakovleva, J. Lafera, W. Baumgärtner, A. Krueger, I. Prinz, A. Gossler, A. Kotlyarov, M. Gaestel

The paper shows that ERK3 is not essential for viability, fertility, or T-cell function, contradicting previous reports of perinatal lethality due to a different genetic construct.

bioRxiv · Nov 10, 2018Cell Biology