Author

Nancy D. Dalton

University of California San Diego

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Genetics· 1 Molecular Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

Jeffrey Y. HuangiD· 2 shared Jeremy Davis‐TurakiD· 2 shared Jon C. NeumanniD· 2 shared Raymond WangiD· 2 shared Shih‐hsin KaniD· 2 shared Yunghang Chan· 2 shared Allisandra K. RhaiD· 1 shared Anthony D. Rangel· 1 shared

Activity by Year

2020-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 34 mutual co-author links

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2 shared papersGenetics

Jeffrey Y. HuangORCID

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Author profile →

3 papers

Recent·Most cited

Lipoxins Regulate Intercalated Disk-Associated Signaling and Immune Remodeling in Dilated Cardiomyopathy

Madison Clark, Kyohei Fujita, Lena Anastasia Magdalena Nielsen, Robert Timothy Johnson, Yusu Gu, Nancy D. Dalton, Bianca E Suur, Ida Bergström, Eric A. Adler, Ju Chen, Marianne Quiding‐Järbrink, Entela Bollano, Niklas Bergh, Matúš Soták, Elisabeth Ehler, , Stephan Lange

This study shows lipoxins modulate immune responses and cardiac signaling in male MLPko mice with dilated cardiomyopathy, but not in females.

bioRxiv · Mar 11, 2026 · PreprintCell Biology

CRISPR-Mediated Generation and Characterization of a Gaa Homozygous c.1935C>A (p.D645E) Pompe Disease Knock-in Mouse Model Recapitulates Human Infantile Onset-Pompe Disease

Shih‐hsin Kan, Jeffrey Y. Huang, Jerry F. Harb, Allisandra K. Rha, Nancy D. Dalton, Chloe L. Christensen, Yunghang Chan, Jeremy Davis‐Turak, Jon C. Neumann, Raymond Wang

This paper generates and characterizes a Gaa c.1935C>A knock-in mouse model of infantile-onset Pompe disease, showing reduced GAA activity, glycogen accumulation, and cardiac hypertrophy.

bioRxiv · May 30, 2022 · PreprintGenetics

CRISPR-Cas9 generated Pompe knock-in murine model exhibits early-onset hypertrophic cardiomyopathy and skeletal muscle weakness

Jeffrey Y. Huang, Shih‐hsin Kan, Emilie K. Sandfeld, Nancy D. Dalton, Anthony D. Rangel, Yunghang Chan, Jeremy Davis‐Turak, Jon C. Neumann, Raymond Wang

This paper describes a CRISPR-Cas9-generated murine model of infantile-onset Pompe Disease that exhibits early-onset hypertrophic cardiomyopathy and skeletal muscle weakness similar to the human condition.

Scientific Reports · Jun 25, 2020Peer-reviewedMolecular Biology