Baicalin ameliorates behavioral and physiological deficits in Gtf2i-duplication mice and modulates synaptic activity in 7q11.23 Duplication Syndrome patient-derived neurons
David Peles, Shai Netser, Natalie Ray, Taghreed Suliman, S. Stern, Shlomo Wagner
This study shows that Baicalin treatment reduces behavioral and physiological deficits in Gtf2i-duplication mice and enhances synaptic activity in 7Dup patient-derived neurons.
bioRxiv · Feb 13, 2026 · PreprintNeuroscience