Author

O.-O. Wolz

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cancer Biology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Approach:experimental assay· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Assay:metagenomics· 1 Assay:proteomics· 1

Frequent Co-Authors

A. N. Weber· 1 shared J. Admard· 1 shared R. Siebert· 1 shared S. Dickhöfer· 1 shared S. Fillinger· 1 shared S. H. Bernhart· 1 shared S. Nahnsen· 1 shared S. Ossowski· 1 shared

Activity by Year

2020-2020 · peak 1/yr

Co-Author Collaboration Network

9 top collaborators · 36 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperCancer Biology

A. N. Weber

Mutual co-authors in cluster

Author profile →

1 paper

Recent·Most cited

The failure of B cells to induce non-canonical MYD88 splice variants correlates with lymphomagenesis via sustained NF-κB signaling

Y. Cardona Gloria, S. H. Bernhart, S. Fillinger, O.-O. Wolz, S. Dickhöfer, J. Admard, S. Ossowski, S. Nahnsen, R. Siebert, A. N. Weber

The paper shows that B cells lack a negative feedback loop that normally limits TLR signaling through MYD88 splice variants, leading to sustained NF-κB activation and lymphomagenesis.

bioRxiv · Jun 18, 2020Cancer Biology