Author

S. Nahnsen

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2 Cancer Biology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 3 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Assay:metagenomics· 2 Approach:benchmark study· 1 Research Focus:benchmarking· 1 Research Focus:cancer & tumor microenvironment· 1

Frequent Co-Authors

C. Ruschil· 2 shared G. Gabernet· 2 shared S. Ossowski· 2 shared A. Langarica Fuentes· 1 shared A. N. Weber· 1 shared A. Peltzer· 1 shared A. Schulze-Selting· 1 shared B. Hemmer· 1 shared

Activity by Year

2020-2022 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 16 mutual co-author links

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2 shared papersImmunology

C. Ruschil

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Comparative multi-OMICS single-cell atlas of five COVID-19 (rAdVV and mRNA) vaccines describe unique and distinct mechanisms of action

Y. Singh, A. Schulze-Selting, G. Gabernet, U. Ray, R. Bajaj, M. Ali, M. Loitz, V. Hammer, E. Buena-Atienza, C. Ruschil, J. Huebner, M. Kowarik, M. salker, N. Casadei, S. Nahnsen, , O. Riess

This study compares the mechanisms of action of five COVID-19 vaccines using multi-omics single-cell data, revealing distinct immune responses and cell-type-specific effects.

bioRxiv · Sep 12, 2022 · PreprintImmunology

Interpretations of microbial community studies are biased by the selected 16S rRNA gene amplicon sequencing pipeline.

D. Straub, N. Blackwell, A. Langarica Fuentes, A. Peltzer, S. Nahnsen, S. Kleindienst

This paper compares 16S rRNA gene amplicon sequencing pipelines to identify biases in microbial community analysis and recommends QIIME2 as the most accurate tool.

Frontiers in Microbiology · Oct 23, 2020Peer-reviewedMicrobiology

Specific induction of double negative B cells during protective and pathogenic immune responses

C. Ruschil, G. Gabernet, G. Lepennetier, S. Heumos, M. Kaminski, Z. Hrasko, M. Irmler, J. Beckers, U. Ziemann, S. Nahnsen, G. P. Owens, J. L. Bennett, B. Hemmer, M. C. Kowarik

The paper identifies double negative B cells as an inducible population expanded in inflammatory diseases and vaccine responses, showing clonal expansion and antigen-specific antibody production.

bioRxiv · Sep 8, 2020Immunology

The failure of B cells to induce non-canonical MYD88 splice variants correlates with lymphomagenesis via sustained NF-κB signaling

Y. Cardona Gloria, S. H. Bernhart, S. Fillinger, O.-O. Wolz, S. Dickhöfer, J. Admard, S. Ossowski, S. Nahnsen, R. Siebert, A. N. Weber

The paper shows that B cells lack a negative feedback loop that normally limits TLR signaling through MYD88 splice variants, leading to sustained NF-κB activation and lymphomagenesis.

bioRxiv · Jun 18, 2020Cancer Biology