Author

P. Knolle

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:human· 2 Research Focus:cell type annotation· 1 Approach:deep learning· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Organism:mouse· 1

Frequent Co-Authors

A. Dueck· 1 shared A. Odainic· 1 shared A. Roers· 1 shared A. Uzonyi· 1 shared A. Welle· 1 shared B. Steglich· 1 shared C. E. Zielinski· 1 shared C. Kurts· 1 shared

Activity by Year

2018-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 12 mutual co-author links

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1 shared paperCell Biology

A. Dueck

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

The lncRNA landscape of cardiac resident macrophages and identification of Schlafenlnc as a regulator of macrophage migratory function

A. Dueck, L. Althaus, K. Heise, D. Esfandyari, S. Bayguen, R. P. Brandes, J. Gagneur, N. Jae, P. Knolle, M. S. Leisegang, L. Maegdefessel, T. Meitinger, D. Niessing, N. Petzold, D. Ramanujam, , S. Engelhardt

The paper identifies lncRNAs in cardiac resident macrophages and shows Schlafenlnc regulates macrophage migration.

bioRxiv · Nov 30, 2022 · PreprintCell Biology

Suppression of systemic T cell immunity to viral infection during liver injury is prevented by inhibition of interferon and IL-10 signaling

P. C. Hackstein, J. Spitzer, K. Symeonidis, H. Horvatic, T. Bedke, B. Steglich, L. Assmus, A. Odainic, N. Kessler, S. Klein, M. Beyer, R. Schmithausen, E. Latz, C. Kurts, J. Trebicka, , Z. Abdullah

The paper shows that liver injury suppresses systemic T cell immunity to viral infection, which can be reversed by inhibiting interferon and IL-10 signaling in preclinical models and human patients.

bioRxiv · Jul 21, 2022 · PreprintImmunology

Sodium chloride is an ionic checkpoint for human Th2 cell responses and shapes the atopic skin microenvironment

J. Matthias, J. Maul, R. Noster, H. Meinl, Y.-Y. Chao, H. Gerstenberg, F. Jeschke, G. Gilles, A. Welle, J. Walter, K. Nordstroem, K. Eberhardt, D. Renisch, S. Donakonda, P. Knolle, , C. E. Zielinski

This study shows sodium chloride promotes Th2 cell responses and shapes the atopic skin microenvironment, suggesting it as a therapeutic target for Th2-mediated diseases.

bioRxiv · Aug 13, 2018Immunology