Author

P. Paganetti

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 3 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Organism:human· 3 Assay:bulk RNA-seq· 2 Research Focus:cell signaling & trafficking· 2 Research Focus:chromatin & epigenetic regulation· 2 Approach:experimental assay· 2 Approach:biophysical modeling· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

S. Papin· 3 shared A. Rinaldi· 2 shared C. Magrin· 2 shared M. Bolis· 2 shared M. Sola· 2 shared A. Clery· 1 shared A. Zbinden· 1 shared C. Foglieni· 1 shared

Activity by Year

2020-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 19 mutual co-author links

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3 shared papersCell Biology

S. Papin

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Author profile →

4 papers

Recent·Most cited

Cancer-specific association between Tau (MAPT) and cellular pathways, clinical outcome, and drug response

M. Callari, M. Sola, C. Magrin, A. Rinaldi, M. Bolis, P. Paganetti, L. Colnaghi, S. Papin

The paper analyzes MAPT expression across cancer types to show its association with cancer hallmarks, clinical outcomes, and drug response, highlighting context-specific roles.

bioRxiv · Jul 4, 2023Cancer Biology

Tau protein modulates an epigenetic mechanism of cellular senescence

C. Magrin, M. Sola, E. Piovesana, M. Bolis, A. Rinaldi, S. Papin, P. Paganetti

The paper investigates how Tau protein affects epigenetic regulation of cellular senescence by analyzing transcriptome data from Tau-depleted cells.

bioRxiv · Jun 5, 2023 · PreprintCell Biology

TDP-43 oligomerization and RNA binding are codependent but their loss elicits distinct pathologies

M. Perez-Berlanga, V. I. Wiersma, A. Zbinden, L. De Vos, U. Wagner, C. Foglieni, I. Mallona, K. M. Betz, A. Clery, J. Weber, Z. Guo, R. Rigort, P. de Rossi, R. Manglunia, E. Tantardini, , M. Polymenidou

This study shows TDP-43 oligomerization and RNA binding work together to control its stability and localization, and their loss leads to different pathological outcomes.

bioRxiv · May 23, 2022 · PreprintCell Biology

Extracellular Vesicles Hijack the Autophagic Pathway to Induce Tau Accumulation in Endolysosomes

G. Pedrioli, M. Barberis, M. Molinari, D. Morone, S. Papin, P. Paganetti

This study shows extracellular vesicles deliver Tau seeds to endolysosomes, where autophagy promotes Tau accumulation and cytotoxicity, suggesting autophagy stimulation may worsen tauopathies.

bioRxiv · May 27, 2020 · PreprintCell Biology