Author

P. Pelczar

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Research Focus:differential expression· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

A. Borchers· 1 shared A. Giannou· 1 shared A. Machicote· 1 shared A. Zimmermann· 1 shared B. Demarco· 1 shared B. Liu· 1 shared B. M. Beckmann· 1 shared B. Steglich· 1 shared

Activity by Year

2021-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 22 mutual co-author links

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1 shared paperImmunology

A. Borchers

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Emergence and suppressive function of Tr1 cells in glomerulonephritis

S. Soukou-Wargalla, C. Kilian, L. Velasquez, A. Machicote, F. Stumme, T. Bedke, A. Giannou, J. Kempski, M. Sabihi, N. Song, H.-J. Paust, A. Borchers, L. G. Perez, P. Pelczar, B. Liu, , S. Huber

This study identifies Tr1 cells in glomerulonephritis, showing their suppressive function and potential as therapeutic targets.

bioRxiv · Feb 7, 2023Immunology

Gasdermin D is the only Gasdermin that provides non-redundant protection against acute Salmonella gut infection

S. A. Fattinger, L. Maurer, P. Geiser, U. Enz, S. Ganguillet, E. Gul, S. Kroon, B. Demarco, M. Furter, M. Barthel, P. Pelczar, F. Shao, P. Broz, M. E. Sellin, W.-D. Hardt

This study identifies Gasdermin D as the only Gasdermin providing non-redundant protection against acute Salmonella Typhimurium gut infection by restricting bacterial loads, controlling inflammation, and preventing epithelial disruption.

bioRxiv · Nov 24, 2022Immunology

Defective Desmosomal Adhesion Causes Arrhythmogenic Cardiomyopathy by involving an Integrin-αVβ6/TGF-β Signaling Cascade

C. Schinner, H. Franz, A. Zimmermann, M.-T. Wanuske, F. Geier, P. Pelczar, V. Lorenz, L. Xu, C. Stuedle, P. I. Maly, S. Kauferstein, B. M. Beckmann, G. M. Kuster, V. Spindler

This study shows that defective desmosomal adhesion causes arrhythmogenic cardiomyopathy through an integrin-αVβ6/TGF-β signaling cascade, leading to fibrosis and arrhythmias.

bioRxiv · Sep 2, 2021Cell Biology