Author

S. A. Fattinger

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 3 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Organism:mouse· 4 Research Focus:host-pathogen & immune response· 3 Research Focus:cell type annotation· 2 Research Focus:cell-cell communication· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:differential expression· 1 Organism:non-human primate· 1

Frequent Co-Authors

W.-D. Hardt· 3 shared M. Barthel· 2 shared M. E. Sellin· 2 shared M. Furter· 2 shared A. Hausmann· 1 shared A. Moon· 1 shared A. Rocker· 1 shared A. Woller· 1 shared

Activity by Year

2022-2023 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 19 mutual co-author links

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3 shared papersImmunology

W.-D. Hardt

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Intraluminal neutrophils limit epithelium damage by reducing pathogen assault on intestinal epithelial cells during Salmonella gut infection

E. Gül, S. A. Fattinger, B. D. Nguyen, A. Hausmann, M. Furter, M. Barthel, M. E. Sellin, W.-D. Hardt

This study shows that intraluminal neutrophils protect the intestinal epithelium from Salmonella-induced damage by reducing pathogen load and physical blockage during infection.

bioRxiv · Feb 23, 2023 · PreprintImmunology

Gasdermin D is the only Gasdermin that provides non-redundant protection against acute Salmonella gut infection

S. A. Fattinger, L. Maurer, P. Geiser, U. Enz, S. Ganguillet, E. Gul, S. Kroon, B. Demarco, M. Furter, M. Barthel, P. Pelczar, F. Shao, P. Broz, M. E. Sellin, W.-D. Hardt

This study identifies Gasdermin D as the only Gasdermin providing non-redundant protection against acute Salmonella Typhimurium gut infection by restricting bacterial loads, controlling inflammation, and preventing epithelial disruption.

bioRxiv · Nov 24, 2022Immunology

Early cellular mechanism of type I interferon-driven susceptibility to tuberculosis

D. I. Kotov, O. V. Lee, S. A. Fattinger, C. Langner, J. V. Guillen, J. M. Peters, A. Moon, E. M. Burd, K. C. Witt, D. B. Stetson, D. L. Jaye, B. D. Bryson, R. E. Vance

This study identifies interstitial macrophages and plasmacytoid dendritic cells as key producers of type I IFN during Mtb infection, showing their role in driving tuberculosis pathogenesis through IFN-driven mechanisms.

bioRxiv · Oct 6, 2022Immunology

Vaccine-enhanced competition permits rational bacterial strain replacement in the gut

V. Lentsch, A. Woller, A. Rocker, S. Aslani, C. Moresi, N. Ruoho, L. Larsson, S. A. Fattinger, N. Wenner, E. Cappio Barazzone, W.-D. Hardt, C. Loverdo, M. Diard, E. Slack

The paper demonstrates that oral vaccination combined with engineered competitor strains can exclude pathogens and replace bacterial strains in the mouse gut.

bioRxiv · Jul 20, 2022Microbiology