Author

Paul J. Vorster

Stanford University

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

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Developmental Biology· 1 Molecular Biology· 1

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Research Focus:differential expression· 2 Research Focus:gene regulatory network inference· 2 Assay:bulk RNA-seq· 1 Organism:C. elegans· 1 Research Focus:cell type annotation· 1 Organism:Drosophila· 1 Approach:experimental assay· 1 Organism:mouse· 1

Frequent Co-Authors

AMANDA J.G. DICKINSONiD· 1 shared Braden J Larson· 1 shared Gregory S. WalshiD· 1 shared John Ojumu· 1 shared Joseph S. LipsickiD· 1 shared Keelan Z. GuileyiD· 1 shared Laura AndrejkaiD· 1 shared Paul D. GoetschiD· 1 shared

Activity by Year

2019-2020 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperDevelopmental Biology

AMANDA J.G. DICKINSONORCID

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2 papers

Recent·Most cited

A Long Lost Key Opens an Ancient Lock: Drosophila Myb Causes a Synthetic Multivulval Phenotype in Nematodes

Paul J. Vorster, Paul D. Goetsch, Tilini U. Wijeratne, Keelan Z. Guiley, Laura Andrejka, Sarvind Mani Tripathi, Braden J Larson, Seth M. Rubin, Susan Strome, Joseph S. Lipsick

This paper shows that Drosophila Myb can bind to nematode LIN9 and LIN52 proteins and cause a synthetic multivulval phenotype in C. elegans, despite their distant evolutionary divergence.

Biology Open · Jan 1, 2020Peer-reviewedMolecular Biology

Loss of Nance-Horan Syndrome b (nhsb) prevents expansion growth of retinal progenitor cells by selective up-regulation of Δ113p53

Paul J. Vorster, John Ojumu, AMANDA J.G. DICKINSON, Gregory S. Walsh

This study shows that loss of Nhs in zebrafish and Xenopus leads to small eye size due to reduced proliferation, linked to increased Δ113p53 and its target genes.

bioRxiv · Oct 1, 2019 · PreprintDevelopmental Biology