Author

R. E. Rau

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:differential expression· 1 Assay:DNA methylation· 1 Approach:experimental assay· 1 Approach:genetic perturbation / CRISPR· 1

Frequent Co-Authors

J. M. Reyes· 2 shared M. A. Goodell· 2 shared A. C. Daquinag· 1 shared A. G. Guzman· 1 shared A. Guzman· 1 shared A. R. Cox· 1 shared A. Tovy· 1 shared C. D. D. Cristobal· 1 shared

Activity by Year

2020-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 30 mutual co-author links

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2 shared papersCell Biology

J. M. Reyes

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Author profile →

2 papers

Recent·Most cited

Constitutive loss of DNMT3A causes morbid obesity through misregulation of adipogenesis

A. Tovy, J. M. Reyes, L. Zhang, Y.-H. Huang, C. Rosas, A. C. Daquinag, A. G. Guzman, R. Ramabadran, C.-W. Chen, T. Gu, S. Gupta, L. Ortinau, D. Park, A. R. Cox, R. E. Rau, , M. A. Goodell

This study shows that loss of DNMT3A causes obesity by disrupting adipogenesis and triggering inflammation in adipose tissue.

eLife · May 30, 2022Peer-reviewedCell Biology

Modeling IKZF1 lesions in B-ALL reveals distinct chemosensitivity patterns and potential therapeutic vulnerabilities

J. H. Rogers, R. Gupta, J. M. Reyes, M. Gundry, G. Medrano, A. Guzman, T. Song, C. Johnson, S. Barnes, C. D. D. Cristobal, L. Brunetti, M. A. Goodell, R. E. Rau

This study models IKZF1 lesions in B-ALL to show distinct chemosensitivity patterns and therapeutic vulnerabilities, identifying cytarabine sensitivity and JAK/STAT upregulation as potential targets.

bioRxiv · May 26, 2020Cancer Biology