Author

R. G. Roeder

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Microbiology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Research Focus:chromatin & epigenetic regulation· 3 Approach:experimental assay· 3 Research Focus:cell type annotation· 2 Organism:mouse· 2 Assay:ATAC-seq· 1 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Assay:ChIP-seq· 1

Frequent Co-Authors

A. Chadburn· 1 shared A. Gerber· 1 shared A. M. Melnick· 1 shared A. Tarakhovsky· 1 shared B. A. Garcia· 1 shared B. D. Dill· 1 shared C. Adura· 1 shared C. E. Mason· 1 shared

Activity by Year

2018-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperCancer Biology

A. Chadburn

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Cooperative super-enhancer inactivation caused by heterozygous loss of CREBBP and KMT2D skews B cell fate decisions and yields T cell-depleted lymphomas

J. Li, C. R. Chin, H.-Y. Ying, C. Meydan, M. R. Teater, M. Xia, P. Farinha, K. Takata, C.-S. Chu, M. A. Rivas, A. Chadburn, C. Steidl, D. W. Scott, R. G. Roeder, C. E. Mason, , A. M. Melnick

This study shows that combined loss of CREBBP and KMT2D disrupts super-enhancer function in B cells, leading to immune evasion and T cell depletion in lymphomas.

bioRxiv · Feb 13, 2023Cancer Biology

A histone-like motif in yellow fever virus contributes to viral replication

D. Mourao, S. Chen, U. Schaefer, L. Bozzacco, L. A. Carneiro, A. Gerber, C. Adura, B. D. Dill, H. Molina, T. Carroll, M. Paul, N. V. Bhanu, B. A. Garcia, G. Joberty, I. Rioja, , A. Tarakhovsky

This study identifies a histone-like motif in the yellow fever virus capsid protein that mimics histone H4 and interferes with BRD4-mediated gene regulation, impacting viral replication.

bioRxiv · May 5, 2020 · PreprintMicrobiology

A non-canonical-PPARγ/RXRα-binding sequence regulates leptin expression in response to changes in adipose tissue mass

Y. Zhang, O. S. Dallner, T. Nakadai, G. Fayzikhodjaeva, Y.-H. Lu, M. A. Lazar, R. G. Roeder, J. M. Friedman

The paper identifies a non-canonical PPARγ/RXRα-binding site that regulates leptin expression in response to adipose tissue mass changes.

bioRxiv · May 18, 2018Physiology