Author

R. Gonzalez-Novo

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Approach:biophysical modeling· 2 Research Focus:cell signaling & trafficking· 2 Research Focus:cell type annotation· 2 Research Focus:chromatin & epigenetic regulation· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Assay:DNA methylation· 1

Frequent Co-Authors

J. Redondo-Munoz· 3 shared A. De Lope-Planelles· 2 shared E. Madrazo· 2 shared F. Monroy· 2 shared H. Lopez-Menendez· 2 shared p. Roda-Navarro· 2 shared A. De Lope· 1 shared A. Gonzalez-Murillo· 1 shared

Activity by Year

2022-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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3 shared papersCell Biology

J. Redondo-Munoz

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3 papers

Recent·Most cited

Cell BiologybioRxivPreprintApr 10, 2023

3D environment promotes persistent changes in lamin B1 distribution, the biomechanical signature of the nucleus, and adaptative survival and migratory functions.

R. Gonzalez-Novo, H. Zamora Carreras, A. De Lope-Planelles…·J. Redondo-Munoz

The paper shows that 3D environments cause persistent nuclear changes, including lamin B1 distribution and mechanical behavior, affecting cell survival and migration.

DOI 10.1101/2023.04.10.536202

experimental assaybiophysical modeling

𝕏 PostBluesky

Cell BiologybioRxivPreprintJan 5, 2023

Persistent confined migration confers permanent nuclear and functional changes in migrating cells.

A. de Lope-Planelles, R. Gonzalez-Novo, E. Madrazo…·J. Redondo-Munoz

The paper shows that persistent confined migration causes permanent nuclear and functional changes in cells, including lamin B1 redistribution and altered DNA repair.

DOI 10.1101/2023.01.05.522838

proteomicsDNA methylation

𝕏 PostBluesky

Cancer BiologybioRxivPreprintJun 16, 2022

Targeting H3K4 methylation as a novel therapeutic strategy against tumor infiltration and nuclear changes of acute lymphoblastic leukemia cells.

R. Gonzalez-Novo, A. De Lope, A. Gonzalez-Murillo…·J. Redondo-Munoz

This study shows that targeting H3K4 methylation via WDR5 inhibition reduces leukemia cell invasion in vitro and tissue infiltration in vivo, revealing a novel therapeutic strategy for acute lymphoblastic leukemia.

DOI 10.1101/2022.06.16.495903

humanbulk RNA-seq

𝕏 PostBluesky