Author

R. J. Cornall

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2 Cell Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 3 Approach:experimental assay· 3 Organism:mouse· 2 Research Focus:cell-cell communication· 1 Research Focus:development & morphogenesis· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1 Research Focus:metabolism & bioenergetics· 1

Frequent Co-Authors

C. Liu· 2 shared A. Chen· 1 shared A. J. Clarke· 1 shared A. J. Mentzer· 1 shared A. K. Simon· 1 shared A. McMichael· 1 shared B. M. Kessler· 1 shared B. Qin· 1 shared

Activity by Year

2017-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 17 mutual co-author links

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2 shared papersCell Biology

C. Liu

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Author profile →

3 papers

Recent·Most cited

Spatiotemporal transcriptomic atlas of mouse organogenesis using DNA nanoball patterned arrays

A. Chen, S. Liao, M. Cheng, K. Ma, L. Wu, Y. Lai, X. Qiu, J. Yang, W. Li, J. Xu, S. Hao, X. Wang, H. Lu, X. Chen, X. Liu, , J. Wang

This study develops Stereo-seq, a high-resolution spatial transcriptomic method, to analyze mouse organogenesis and map developmental disease loci.

Cell · May 1, 2022Peer-reviewedCell Biology

Broad and strong memory CD4+ and CD8+ T cells induced by SARS-CoV-2 in UK convalescent COVID-19 patients.

T. Dong, Y. Peng, A. J. Mentzer, G. Liu, X. Yao, Z. Yin, D. Dong, W. Dejnirattisai, L. Turtle, T. Rostron, K. Subramaniam, P. Thomson, P. Zhang, C. Dold, J. Ratcliff, , Lopez-C

This study examines T cell immunity in UK COVID-19 patients, finding stronger and broader memory T cell responses in severe cases, with implications for vaccine design.

bioRxiv · Jun 5, 2020Immunology

B1a B cells require autophagy for metabolic homeostasis and self-renewal

A. J. Clarke, T. Riffelmacher, D. Braas, R. J. Cornall, A. K. Simon

This study shows B1a B cells rely on autophagy for metabolic homeostasis and self-renewal, with Atg7 deletion causing their loss due to metabolic dysregulation and mitochondrial dysfunction.

bioRxiv · Dec 28, 2017Immunology