Author

R. Ruiz-Vega

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cancer Biology· 2

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:cell type annotation· 2 Organism:mouse· 2 Assay:scRNA-seq· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1

Frequent Co-Authors

A. D. Lander· 2 shared A. K. Ganesan· 2 shared C.-F. Chen· 2 shared J. Shiu· 2 shared E. Razzak· 1 shared H. Xiao· 1 shared H. Yan· 1 shared J. Lowengrub· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersCancer Biology

A. D. Lander

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Cancer BiologybioRxivDec 8, 2023

Uncovering Minimal Pathways in Melanoma Initiation

H. Xiao, J. Shiu, C.-F. Chen…·A. K. Ganesan

This study identifies LNM cells as a pre-malignant state in melanoma initiation, showing they pre-exist in normal skin, expand with Braf activation, and contribute to drug resistance.

DOI 10.1038/s41467-025-60742-0

mousescRNA-seq

𝕏 PostBluesky

Cancer BiologybioRxivJul 14, 2020

Dynamics of nevus development implicate cell cooperation in the growth arrest of transformed melanocytes

R. Ruiz-Vega, C.-F. Chen, E. Razzak…·A. D. Lander

The paper investigates nevus growth arrest using a mouse model and finds that collective cell behavior, not cell-autonomous senescence, explains nevus size control.

DOI 10.7554/elife.61026

mousescRNA-seq

𝕏 PostBluesky