Author

Reed T. Bjork

University of Arizona

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:cell type annotation· 1 Organism:Drosophila· 1 Assay:electronic health records· 1 Organism:zebrafish· 1

Frequent Co-Authors

Alexander D. Blythe· 1 shared Allison F. MichaeliD· 1 shared Brian Dominguez· 1 shared Brijesh Singh ChauhaniD· 1 shared Christi Williams· 1 shared Daniela C. ZarnescuiD· 1 shared Eric B. AlsopiD· 1 shared Gabriel Holguin· 1 shared

Activity by Year

2022-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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1 shared paperNeuroscience

Alexander D. Blythe

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2 papers

Recent·Most cited

Alpha-synuclein overexpression reduces neural activity within a basal ganglia vocal nucleus in a zebra finch model

Brian Dominguez, Gabriel Holguin, Madeleine S. Daly, Reed T. Bjork, Stephen L. Cowen, Julie E. Miller

This study shows that overexpressing alpha-synuclein in zebra finch Area X reduces firing rates of specific neurons, mimicking Parkinsonian-like vocal impairments.

PLoS ONE · Jul 16, 2026Peer-reviewedNeuroscience

Modelling dementia in Drosophila uncovers shared and specific targets of TDP-43 proteinopathy across ALS and FTD relevant circuits

Rebekah Keating Godfrey, Reed T. Bjork, Eric B. Alsop, Brijesh Singh Chauhan, Hillary C. Ruvalcaba, Jerry Antone, Allison F. Michael, Christi Williams, Grace Hala’ufia, Alexander D. Blythe, Megan C. Hall, Kendall R. Van Keuren‐Jensen, Daniela C. Zarnescu

This study uses a Drosophila model to investigate TDP-43 proteinopathy in ALS and FTD, identifying shared and circuit-specific molecular targets.

bioRxiv · Nov 16, 2022 · PreprintNeuroscience