Author

S. Feliziani

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Microbiology· 2

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 2 Organism:human· 2 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:host-pathogen & immune response· 1 Assay:whole-genome sequencing· 1

Frequent Co-Authors

A. M. Smania· 2 shared C. A. Colque· 2 shared H. K. Johansen· 2 shared S. Molin· 2 shared A. Albarracin Orio· 1 shared A. G. Albarracin Orio· 1 shared A. J. Vila· 1 shared A. M. Moyano· 1 shared

Activity by Year

2019-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 40 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersMicrobiology

A. M. Smania

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Development of antibiotic resistance reveals diverse evolutionary pathways to face the complex and dynamic environment of a long-term treated patient

C. A. Colque, P. E. Tomatis, A. G. Albarracin Orio, G. Dotta, D. M. Moreno, L. G. Hedemann, R. A. Hickman, L. M. Sommer, S. Feliziani, A. M. Moyano, R. A. Bonomo, H. K. Johansen, S. Molin, A. J. Vila, A. M. Smania

This study tracks the long-term evolution of antibiotic-resistant Pseudomonas aeruginosa in a cystic fibrosis patient, revealing diverse genetic changes that enhance resistance to multiple cephalosporins.

bioRxiv · May 14, 2021 · PreprintMicrobiology

Hypermutator Pseudomonas aeruginosa exploits multiple genetic pathways to develop multidrug resistance during long term infections in the airways of cystic fibrosis patients

C. A. Colque, A. Albarracin Orio, S. Feliziani, R. L. Marvig, R. A. Tobares, H. K. Johansen, S. Molin, A. M. Smania

This study shows how hypermutator Pseudomonas aeruginosa strains develop multidrug resistance through parallel evolution of distinct genetic pathways during chronic cystic fibrosis infections.

bioRxiv · Oct 17, 2019Microbiology