Author

A. M. Smania

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 2 Organism:human· 2 Organism:bacteria (other)· 1 Research Focus:chromatin & epigenetic regulation· 1 Assay:metabolomics· 1 Assay:whole-genome sequencing· 1

Frequent Co-Authors

A. G. Albarracin Orio· 2 shared C. A. Colque· 2 shared H. K. Johansen· 2 shared R. A. Tobares· 2 shared S. Feliziani· 2 shared S. Molin· 2 shared A. Aksenov· 1 shared A. Albarracin Orio· 1 shared

Activity by Year

2019-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 30 mutual co-author links

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2 shared papersMicrobiology

A. G. Albarracin Orio

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3 papers

Recent·Most cited

Development of antibiotic resistance reveals diverse evolutionary pathways to face the complex and dynamic environment of a long-term treated patient

C. A. Colque, P. E. Tomatis, A. G. Albarracin Orio, G. Dotta, D. M. Moreno, L. G. Hedemann, R. A. Hickman, L. M. Sommer, S. Feliziani, A. M. Moyano, R. A. Bonomo, H. K. Johansen, S. Molin, A. J. Vila, A. M. Smania

This study tracks the long-term evolution of antibiotic-resistant Pseudomonas aeruginosa in a cystic fibrosis patient, revealing diverse genetic changes that enhance resistance to multiple cephalosporins.

bioRxiv · May 14, 2021 · PreprintMicrobiology

Fungal-bacterial interaction selects for quorum sensing mutants and a metabolic shift towards the production of natural antifungal compounds

A. G. Albarracin Orio, D. Petras, R. A. Tobares, A. Aksenov, M. Wang, F. Juncosa, P. Sayago, A. J. Moyano, P. C. Dorrestein, A. M. Smania, D. Ducasse

The paper shows that B. subtilis mutants with quorum-sensing mutations produce antifungal ketones instead of surfactins when interacting with the fungus S. terrestris.

bioRxiv · Jan 16, 2020 · PreprintMicrobiology

Hypermutator Pseudomonas aeruginosa exploits multiple genetic pathways to develop multidrug resistance during long term infections in the airways of cystic fibrosis patients

C. A. Colque, A. Albarracin Orio, S. Feliziani, R. L. Marvig, R. A. Tobares, H. K. Johansen, S. Molin, A. M. Smania

This study shows how hypermutator Pseudomonas aeruginosa strains develop multidrug resistance through parallel evolution of distinct genetic pathways during chronic cystic fibrosis infections.

bioRxiv · Oct 17, 2019Microbiology