Author

S. O. Boe

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biophysics· 1 Cancer Biology· 1 Cell Biology· 1 Pathology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Research Focus:cell type annotation· 3 Research Focus:cell signaling & trafficking· 2 Research Focus:chromatin & epigenetic regulation· 2 Research Focus:development & morphogenesis· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1

Frequent Co-Authors

A. Lang· 3 shared A. Klungland· 2 shared E. Lang· 2 shared J. E. Rinholm· 2 shared A. Anand· 1 shared A. Carlson· 1 shared A. Datta· 1 shared A. Kusnierczyk· 1 shared

Activity by Year

2021-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 26 mutual co-author links

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3 shared papersCancer Biology

A. Lang

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

CDK12/CDK13 inhibition disrupts a transcriptional program critical for glioblastoma survival

S. Lier, S. O. Lund, A. Lipsa, K. B. M. Frauenknecht, I. D. Rein, P. Jain, A. Lang, E. Lang, N. Meyer, A. Datta, S. Anand, G. Nesse, R. J. Forstrom, A. Klungland, J. E. Rinholm, , D. P. Pandey

This study shows that inhibiting CDK12 and CDK13 reduces glioma cell proliferation and migration, and disrupts transcription and cell cycle progression in glioblastoma.

bioRxiv · Jul 14, 2023Cancer Biology

Loss-of-function mutation in human Oxidation Resistance gene 1 disrupts the spatial-temporal regulation of histone arginine methylation in early brain development

X. Lin, W. Wang, M. Yang, N. Damseh, M. M. L. d. Sousa, F. Jacob, A. Lang, E. Kristiansen, M. Pannone, M. Kissova, R. Almaas, A. Kusnierczyk, R. Siller, M. Shahrour, M. Al-Ashhab, , M. Bjoras

This study identifies a loss-of-function mutation in OXR1 causing neurodevelopmental disorders and shows its role in regulating histone arginine methylation in brain organoids.

bioRxiv · May 24, 2022 · PreprintPathology

Mechanical coupling of supracellular stress amplification and tissue fluidization during exit from quiescence

E. Lang, C. Pedersen, A. Lang, P. Blicher, A. Klungland, A. Carlson, S. O. Boe

The paper shows that quiescent human keratinocyte monolayers undergo stress amplification and fluidization upon serum stimulation, linking mechanical stress to tissue motility.

bioRxiv · Oct 19, 2021Biophysics

Kel1 is a phosphorylation-regulated noise suppressor of the pheromone signaling pathway

I. Garcia, S. Munoz, L. Ramos-Alonso, A. N. Andersen, C. Zimmermann, J. Eriksson, S. O. Boe, P. Kaferle, M. Papamichos-Chronakis, P. Chymkowitch, J. Enserink

The paper identifies Kel1 as a phosphorylation-regulated noise suppressor in the yeast pheromone signaling pathway that prevents cell death and spontaneous activation.

bioRxiv · May 19, 2021Cell Biology