Author

S. W. Schultz

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biophysics· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:chromatin & epigenetic regulation· 1 Assay:electronic health records· 1 Approach:experimental assay· 1 Organism:human· 1 Approach:microscopy & imaging analysis· 1 Approach:statistical model· 1

Frequent Co-Authors

A. Brech· 2 shared C. Raiborg· 2 shared H. Stenmark· 2 shared A. Bellanger· 1 shared A. Carlson· 1 shared A. Jain· 1 shared C. Campsteijn· 1 shared C. M. Jones· 1 shared

Activity by Year

2019-2020 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 39 mutual co-author links

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2 shared papersBiophysics

A. Brech

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Protein Crowding Mediates Membrane Remodeling in Upstream ESCRT-induced Formation of Intraluminal Vesicles

S. Liese, E. M. Wenzel, I. Kjos, R. V. Rojas Molina, S. W. Schultz, A. Brech, H. Stenmark, C. Raiborg, A. Carlson

This study shows upstream ESCRTs mediate membrane deformation and ILV formation through crowding and altering bending rigidity, without requiring ATP.

Proceedings of the National Academy of Sciences · Nov 2, 2020Peer-reviewedBiophysics

Unrestrained ESCRT-III drives chromosome fragmentation and micronuclear catastrophe

M. Vietri, S. W. Schultz, A. Bellanger, C. M. Jones, C. Raiborg, E. Skarpen, C. R. J. Pedurupillay, E. Kip, R. Timmer, A. Jain, P. Collas, R. L. Knorr, S. N. Grellscheid, H. Kusumaatmaja, A. Brech, , C. Campsteijn

This study shows that unrestrained ESCRT-III activity at micronuclei causes chromosome fragmentation and DNA damage, while it facilitates repair in primary nuclei.

bioRxiv · Jan 10, 2019Cell Biology