USP39 promotes hepatocellular carcinogenesis through regulating alternative splicing in cooperation with SRSF6/HNRNPC
J. Zheng, S. Wu, M. Tang, S. Xi, Y. Wang, J. Ren, H. Luo, P. Hu, L. Sun, Y. Du, H. Yang, F. Wang, H. Gao, Z. Dai, X. Ou, Y. Li
This study shows USP39 promotes liver cancer by regulating alternative splicing through interaction with SRSF6/HNRNPC, identifying it as a potential therapeutic target.
bioRxiv · Jan 24, 2023 · PreprintCancer Biology