Author

Sammy Pardo

The University of Texas at San Antonio

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Neuroscience· 1

Signature Assays, Organisms & Methods

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Research Focus:differential expression· 2 Assay:proteomics· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1 Assay:metabolomics· 1

Frequent Co-Authors

Susan T. WeintraubiD· 2 shared Ann Marie StoweiD· 1 shared Ann V. GriffithiD· 1 shared Bess E FrostiD· 1 shared Claira SohniD· 1 shared Dana Molleur· 1 shared Eduardo Gutierrez KuriiD· 1 shared Erich J. SohniD· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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Curved chords = mutual co-authorship

2 shared papersNeuroscience

Susan T. WeintraubORCID

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

PLCγ2 deficiency compromises systemic immune tolerance and erodes myelin homeostasis while enhancing oxidative metabolism in the mouse brain

Eduardo Gutierrez Kuri, Juliet Lynn Marie Garcia-Rogers, Jaid Perez, Sabrina Smith, Matthew R. Kenwood, Kahealani S. Archuleta, Yangming Xiao, Gabriela Campos, Savannah Barannikov, Hu Wang, Sammy Pardo, Trevor B. Romsdahl, Henry Miller, Ann Marie Stowe, Russell William, , Juan Pablo Palavicini

This study investigates the effects of PLCγ2 deficiency in mice, showing impaired immune tolerance, myelin homeostasis disruption, and enhanced oxidative metabolism in the brain.

bioRxiv · Jul 14, 2026 · PreprintNeuroscience

Elevation of the mechanically-sensitive protein emerin links nuclear mechanotransduction to tau-induced cytoskeletal remodeling in neurons

Claira Sohn, Sammy Pardo, Dana Molleur, Satvik R Paduri, Morgan Lambert, Zachary Uttke, Erich J. Sohn, Morgan G. Thomas, Susan T. Weintraub, Bess E Frost

This study identifies emerin as a mediator of cytoskeletal remodeling in tauopathy by showing its elevation and relocalization in neuronal models of tauopathy, linking nuclear mechanotransduction to neurotoxicity.

Nucleus · Jul 7, 2026Peer-reviewedCell Biology