Author

Sean R. Duffy

University at Buffalo, State University of New York

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 2 Bioengineering· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:C. elegans· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1

Frequent Co-Authors

John C. PanepintoiD· 2 shared Amanda L. M. BloomiD· 1 shared Anna A. BirukovaiD· 1 shared David GoichiD· 1 shared Kamoltip Promnares· 1 shared Konstantin G. BirukoviD· 1 shared Maritza N. Ventura· 1 shared Pratap KarkiiD· 1 shared

Activity by Year

2024-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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2 shared papersMicrobiology

John C. PanepintoORCID

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Author profile →

3 papers

Recent·Most cited

NMD-mediated control of Tor influences adaptation to nutrient and temperature conditions in Cryptococcus neoformans

Sean R. Duffy, John C. Panepinto

This study shows NMD regulates Tor signaling in C. neoformans, affecting adaptation to nutrient and temperature conditions and fluconazole sensitivity.

bioRxiv · Feb 20, 2026 · PreprintMicrobiology

Adrenomedullin-RAMP2 enhances endothelial cell homeostasis synergistically with shear stress

Yongdae Yoon, Sean R. Duffy, Shannon Elise Kirk, Kamoltip Promnares, Pratap Karki, Anna A. Birukova, Konstantin G. Birukov, Yifan Yuan

The paper identifies ADM/RAMP2 signaling as enhancing endothelial cell homeostasis and barrier integrity, synergistically with shear stress, using in vitro models and functional assays.

Cells · Jan 14, 2026Peer-reviewedBioengineering

Gcn2 rescues reprogramming in the absence of Hog1/p38 signaling in C. neoformans during thermal stress.

David Goich, Amanda L. M. Bloom, Sean R. Duffy, Maritza N. Ventura, John C. Panepinto

This study shows that Gcn2 compensates for Hog1/p38 loss in C. neoformans during thermal stress, rescuing adaptation and virulence through crosstalk between these pathways.

mBio · Dec 13, 2024Peer-reviewedMicrobiology