Author

Shea Sundali

Washington University in St. Louis

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Research Focus:drug response / drug discovery· 2 Approach:experimental assay· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1 Approach:statistical model· 1

Frequent Co-Authors

Aaron DiAntonioiD· 2 shared Jeffrey MilbrandtiD· 2 shared A Joseph Bloom· 1 shared A. Joseph BloomiD· 1 shared Aaron D. GitleriD· 1 shared Amy M. StricklandiD· 1 shared Anushka Bhat· 1 shared Caiwei GuoiD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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2 shared papersNeuroscience

Aaron DiAntonioORCID

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2 papers

Recent·Most cited

Mitochondrial NAD+ import gates neuronal resilience to active SARM1

Lilianne Barbar, Anushka Bhat, Si Jia Chen, Amy M. Strickland, Sarah Schurr, Shea Sundali, Hayoung Kim, Yo Sasaki, A Joseph Bloom, Jeffrey Milbrandt, Aaron DiAntonio

This study shows that enhancing mitochondrial NAD+ import through SLC25A51 protects neurons from SARM1-driven degeneration by preventing coupled metabolic failure.

bioRxiv · Oct 11, 2026 · PreprintNeuroscience

TDP-43 suppression of ATP8A2 cryptic splicing implicates phosphatidylserine-driven neuroinflammation in ALS/FTD

James T. O’Connor, Hui Qi Loo, Caiwei Guo, Sarah R. Pickles, Shea Sundali, Vidhya Maheswari Jawahar, Dennis W. Dickson, A. Joseph Bloom, Leonard Petrucelli, Aaron D. Gitler, Jeffrey Milbrandt, Aaron DiAntonio

This study identifies ATP8A2 as a TDP-43 target whose loss leads to PS exposure and neuroinflammation, suggesting immune cell inhibition as a therapeutic strategy for ALS-FTD and CAMRQ4.

bioRxiv · Nov 21, 2025 · PreprintNeuroscience