Author

T. J. Edwards

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Research Focus:host-pathogen & immune response· 1 Assay:metabolomics· 1 Assay:proteomics· 1 Research Focus:variant effect prediction· 1

Frequent Co-Authors

A. Hopf-Jannasch· 1 shared B. Graham· 1 shared C. Teng· 1 shared D. J. LaCount· 1 shared E. Lian· 1 shared J. L. Edwards· 1 shared J. T. Belisle· 1 shared K. Krieger· 1 shared

Activity by Year

2022-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

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1 shared paperMicrobiology

A. Hopf-Jannasch

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2 papers

Recent·Most cited

Physiological Levels of Estrogen and Progesterone Modulate CR3 expression on, and Neisseria gonorrhoeae Infection of, Primary, Human, Cervical Epithelial Cells

T. J. Edwards, J. L. Edwards

This study shows that physiological estrogen and progesterone levels affect CR3 expression and Neisseria gonorrhoeae infection of cervical cells by altering complement receptor recruitment and Opa protein expression.

bioRxiv · Jun 3, 2022 · PreprintMicrobiology

Shifting a Cellular Metabolic Landscape Identifies a Refractory Environment for Flavivirus Replication

R. C. Gullberg, N. Chotiwan, M. N. Islam, L. St Clair, E. Lian, T. J. Edwards, S. Khadka, C. Teng, B. Graham, K. Krieger, A. Hopf-Jannasch, D. J. LaCount, J. T. Belisle, R. J. Kuhn, R. Perera

The paper identifies a refractory metabolic environment induced by fatty acid biosynthesis inhibition that limits flavivirus replication and shows that combining this with nucleotide synthesis pathway inhibition significantly reduces viral infectivity.

bioRxiv · Feb 21, 2022 · PreprintMicrobiology