Author

T. L. Ivanco

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Organism:mouse· 1 Assay:proteomics· 1 Organism:rat· 1

Frequent Co-Authors

D. Chapman· 2 shared G. M. Hatch· 2 shared J. T. Field· 2 shared J. W. Gordon· 2 shared M. D. Martens· 2 shared A. R. West· 1 shared C. Day· 1 shared C. R. Doucette· 1 shared

Activity by Year

2018-2020 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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2 shared papersCell Biology

D. Chapman

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2 papers

Recent·Most cited

Misoprostol Attenuates Cardiomyocyte Proliferation in the Neonatal Heart Through Bnip3 and Perinuclear Calcium Signaling

M. D. Martens, J. T. Field, N. Seshadri, C. Day, D. Chapman, R. Keijzer, C. R. Doucette, G. M. Hatch, A. R. West, T. L. Ivanco, J. W. Gordon

This study shows misoprostol reduces neonatal cardiomyocyte proliferation via Bnip3 and calcium signaling, offering potential therapeutic benefits for neonatal heart conditions.

Journal of Molecular and Cellular Cardiology · Sep 1, 2020Peer-reviewedCell Biology

Misoprostol regulates Bnip3 repression and alternative splicing to control cellular calcium homeostasis during hypoxic stress.

J. T. Field, M. D. Martens, W. Mughal, Y. Hai, D. Chapman, G. M. Hatch, T. L. Ivanco, W. Diehl-Jones, J. W. Gordon

This study shows misoprostol reduces Bnip3 expression and promotes alternative splicing to protect cells from hypoxia-induced damage by regulating calcium homeostasis.

bioRxiv · May 2, 2018Cell Biology