Author

T. Sugawa

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Ecology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Research Focus:host-pathogen & immune response· 1 Assay:proteomics· 1

Frequent Co-Authors

I. Yamauchi· 3 shared N. Inagaki· 3 shared T. Hakata· 3 shared Y. Ueda· 3 shared D. Kosugi· 2 shared D. Taura· 2 shared H. Fujita· 2 shared K. Okamoto· 2 shared

Activity by Year

2022-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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3 shared papersCell Biology

I. Yamauchi

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

High-throughput screening for Cushing's disease: therapeutic potential of thiostrepton via cell cycle regulation

T. Hakata, I. Yamauchi, D. Kosugi, T. Sugawa, H. Fujita, K. Okamoto, Y. Ueda, T. Fujii, D. Taura, N. Inagaki

This study identifies thiostrepton as a potential therapy for Cushing's disease by inducing cell cycle arrest in PitNETs through mechanisms independent of FOXM1.

bioRxiv · Feb 22, 2024Cell Biology

Transcriptomic Landscape of Hyperthyroidism in Mice Overexpressing Thyroid Stimulating Hormone

I. Yamauchi, T. Sugawa, T. Hakata, A. Yoshizawa, T. Kita, Y. Kishimoto, S. Kimura, D. Kosugi, H. Fujita, K. Okamoto, Y. Ueda, T. Fujii, D. Taura, Y. Sakane, A. Yasoda, N. Inagaki

This study analyzes the transcriptome of hyperthyroid mice overexpressing TSH, identifying novel genes and pathways associated with goiter development and thyroid hormone secretion.

bioRxiv · Oct 27, 2023Physiology

TRIAC disrupts cerebral thyroid hormone action via a negative feedback loop and heterogenous distribution among organs

I. Yamauchi, T. Hakata, Y. Ueda, T. Sugawa, R. Omagari, Y. Teramoto, S. F. Nakayama, D. Nakajima, T. Kubo, N. Inagaki

This study shows TRIAC disrupts cerebral thyroid hormone action by reducing circulating TH levels and being poorly distributed to the brain, while compensating in peripheral tissues.

bioRxiv · Aug 12, 2022Ecology