Author

Tim Holler

Medizinische Hochschule Hannover

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biophysics· 1

Signature Assays, Organisms & Methods

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Research Focus:chromatin & epigenetic regulation· 1 Approach:experimental assay· 1 Organism:human· 1 Approach:mechanistic / dynamical model· 1 Approach:statistical model· 1

Frequent Co-Authors

Arnab NayakiD· 1 shared Cristobal G. dos RemediosiD· 1 shared Emrulla SpahiuiD· 1 shared Kenneth S. CampbelliD· 1 shared Mamta Amrute‐NayakiD· 1 shared Michael RegnieriD· 1 shared Theresia KraftiD· 1 shared Thomas ThumiD· 1 shared

Activity by Year

2026-2026 · peak 1/yr

Co-Author Collaboration Network

9 top collaborators · 36 mutual co-author links

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1 shared paperBiophysics

Arnab NayakORCID

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1 paper

Recent·Most cited

Single-molecule analysis sheds light on cardiac myosin dysfunction due to hypertrophic cardiomyopathy mutation A57D in ventricular myosin light chain-1 (MLC1v)

Tianbang Wang, Emrulla Spahiu, Tim Holler, Kenneth S. Campbell, Cristobal G. dos Remedios, Thomas Thum, Theresia Kraft, Michael Regnier, Arnab Nayak, Mamta Amrute‐Nayak

This study investigates the molecular mechanism of cardiac myosin dysfunction caused by the A57D mutation in MLC1v using single-molecule analysis and computational modeling.

bioRxiv · Mar 9, 2026 · PreprintBiophysics