Author

Emrulla Spahiu

Medizinische Hochschule HannoverORCID 0000-0003-3251-026XUnclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Biophysics· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:chromatin & epigenetic regulation· 1 Assay:fluorescence / live-cell microscopy· 1 Approach:mechanistic / dynamical model· 1 Approach:statistical model· 1

Frequent Co-Authors

Arnab NayakiD· 2 shared Mamta Amrute‐NayakiD· 2 shared Theresia KraftiD· 2 shared Tianbang WangiD· 2 shared Albin E. Berg· 1 shared Alf MånssoniD· 1 shared Cristobal dos Remedios· 1 shared Cristobal G. dos RemediosiD· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

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2 shared papersBiophysics

Arnab NayakORCID

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2 papers

Recent·Most cited

BiophysicsbioRxivPreprintSep 16, 2026

Myotropes unveil two myosin cycles with distinct kinetics and stroke size

Albin E. Berg, Tianbang Wang, Emrulla Spahiu…·Mamta Amrute‐Nayak

The paper uses single-molecule techniques to reveal two distinct myosin cycles with different kinetics and stroke sizes affected by mavacamten and omecamtiv mecarbil.

DOI 10.64898/2026.09.11.750962

humanfluorescence / live-cell microscopy

𝕏 PostBluesky

BiophysicsbioRxivPreprintMar 9, 2026

Single-molecule analysis sheds light on cardiac myosin dysfunction due to hypertrophic cardiomyopathy mutation A57D in ventricular myosin light chain-1 (MLC1v)

Tianbang Wang, Emrulla Spahiu, Tim Holler…·Mamta Amrute‐Nayak

This study investigates the molecular mechanism of cardiac myosin dysfunction caused by the A57D mutation in MLC1v using single-molecule analysis and computational modeling.

DOI 10.64898/2026.03.06.710098

humanexperimental assay

𝕏 PostBluesky