U. Reichl

Detection, isolation and characterisation of phage-host complexes using BONCAT and click chemistry

This paper develops a workflow using BONCAT and click chemistry to detect and isolate phage-host complexes, validated with E. coli and lambda phage.

Macrophage- and CD4+ T cell-derived SIV differ in glycosylation, infectivity and neutralization sensitivity

This study compares glycosylation, infectivity, and neutralization sensitivity of SIV produced in macrophages versus CD4+ T cells, revealing differences in glycan profiles and their impact on viral spread and antibody neutralization.

Semi-continuous propagation of influenza A virus and its defective interfering particles: analyzing the dynamic competition to select candidates for antiviral therapy

This study investigates the competition between influenza A virus and its defective interfering particles (DIPs) during co-infection, identifying DIPs with superior antiviral efficacy as potential therapeutic candidates.

Cell culture-based production of defective interfering influenza A virus particles in perfusion mode using an alternating tangential flow filtration system

This paper establishes an automated perfusion process for producing IAV DIPs using an alternating tangential flow filtration system, achieving high DIP titers and enabling continuous virus harvesting.

Integrated cycles for urban biomass as a strategy to promote a CO2-neutral society - A feasibility study

This paper evaluates the feasibility of integrating closed biomass cycles into residential buildings to achieve CO2 neutrality, showing technical viability but higher costs compared to revenues.

Critical Assessment of Metaproteome Investigation (CAMPI): a Multi-Lab Comparison of Established Workflows

CAMPI evaluates metaproteomic workflows across labs, finding sample processing drives peptide variability while workflows yield similar functional profiles.

Antiviral activity of influenza A virus defective interfering particles against SARS-CoV-2 replication in vitro through stimulation of innate immunity

This study shows that influenza A virus defective interfering particles inhibit SARS-CoV-2 replication in vitro by enhancing innate immunity through JAK/STAT signaling.