Author

V. C. Young

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biophysics· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Assay:clinical cohort / trial· 1 Research Focus:differential expression· 1 Approach:genetic perturbation / CRISPR· 1 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

A. Katz· 1 shared A. Padro· 1 shared B. J. Baker· 1 shared C. M. Wilczewski· 1 shared C. Turner· 1 shared F. Rossignol· 1 shared G. Maxwell· 1 shared G. Mueller· 1 shared

Activity by Year

2023-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperPhysiology

A. Katz

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Author profile →

2 papers

Recent·Most cited

S1 hydrophobic residues modulate voltage sensing phosphatase enzymatic function and voltage sensing

V. Rayaprolu, H. M. Miettinen, W. Baker, V. C. Young, M. Fisher, G. Mueller, W. O. Rankin, J. J. Kelley, W. Ratzan, L. M. Leong, J. A. Davisson, B. J. Baker, S. C. Kohout

This study shows that S1 hydrophobic residues in voltage sensing phosphatase (VSP) modulate voltage sensing and enzymatic activity by altering voltage dependence and kinetics.

bioRxiv · Dec 27, 2023Biophysics

ATP1A1-linked diseases require a malfunctioning protein product from one allele

K. Spontarelli, V. C. Young, R. Sweazey, A. Padro, J. Lee, T. Bueso, R. M. Hernandez, J. Kim, A. Katz, F. Rossignol, C. Turner, C. M. Wilczewski, G. Maxwell, M. Holmgren, J. D. Bailoo, , P. Artigas

The paper investigates whether haploinsufficiency from protein-null ATP1A1 variants causes disease, finding that Atp1a1+/- mice are phenotypically normal and a human with a null variant shows no disease features.

bioRxiv · Mar 5, 2023Physiology