Author

V. Morris

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 1 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Research Focus:cell type annotation· 1 Research Focus:clustering· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Research Focus:protein folding & dynamics· 1 Assay:protein structure· 1

Frequent Co-Authors

A. K. Shalek· 1 shared B. Reif· 1 shared C. Goebl· 1 shared C. Hartlmueller· 1 shared E. Lummertz da Rocha· 1 shared G. Q. Daley· 1 shared G. Rowe· 1 shared H. de Ruiter· 1 shared

Activity by Year

2019-2020 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperCancer Biology

A. K. Shalek

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Author profile →

2 papers

Recent·Most cited

Single-cell analysis reveals mechanisms of plasticity of leukemia initiating cells

V. Morris, W. Marion, T. Hughes, P. Sousa, P. Sensharma, Y. Pikman, M. Harris, A. K. Shalek, T. North, G. Q. Daley, E. Lummertz da Rocha, G. Rowe

The paper uses single-cell transcriptomics to show that leukemia initiating cells can convert between states, with MYC-driven oxidative phosphorylation controlling this plasticity.

bioRxiv · Apr 29, 2020 · PreprintCancer Biology

Cysteine oxidation triggers amyloid fibril formation of the tumor suppressor p16INK4A

C. Goebl, V. Morris, L. van Dam, M. Visscher, P. Polderman, C. Hartlmueller, H. de Ruiter, M. Hora, L. Liesinger, R. Birner-Gruenberger, H. Vos, B. Reif, T. Madl, T. Dansen

This study shows that cysteine oxidation in p16INK4A leads to amyloid fibril formation, which inactivates its tumor suppressor function.

bioRxiv · Jan 2, 2019Biochemistry