Author

V. V. Polosukhin

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cell Biology· 1 Pathology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:mouse· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:development & morphogenesis· 1

Frequent Co-Authors

D. S. Nichols· 2 shared J. J. Gokey· 2 shared S. S. Gutor· 2 shared T. S. Blackwell· 2 shared W. Han· 2 shared A. E. Dikalova· 1 shared A. S. McCall· 1 shared B. W. Richmond· 1 shared

Activity by Year

2023-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 39 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersCell Biology

D. S. Nichols

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Epithelial Yap/Taz are required for functional alveolar regeneration following acute lung injury

G. T. DiGiovanni, W. Han, T. Sherrill, C. J. Taylor, D. S. Nichols, N. M. Geis, U. K. Singha, C. L. Calvi, A. S. McCall, M. M. Dixon, Y. Lui, J.-H. Jang, S. S. Gutor, V. V. Polosukhin, T. S. Blackwell, , J. J. Gokey

This study shows that Yap/Taz in AT2 cells are essential for alveolar regeneration and preventing fibrosis after lung injury.

bioRxiv · Jun 22, 2023Cell Biology

Repetitive Sulfur Dioxide Exposure in Mice Models Post-Deployment Respiratory Syndrome

S. S. Gutor, R. I. Salinas, D. S. Nichols, J. M. R. Bazzano, W. Han, J. J. Gokey, G. Vasiukov, J. D. West, D. C. Newcomb, A. E. Dikalova, B. W. Richmond, S. I. Dikalov, T. S. Blackwell, V. V. Polosukhin

This study develops a mouse model of repetitive sulfur dioxide exposure that mimics post-deployment respiratory syndrome features, identifying oxidative stress as a mediator of pulmonary vascular disease.

bioRxiv · May 15, 2023Pathology