Author

T. S. Blackwell

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 5 Pathology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 6 Organism:mouse· 5 Research Focus:cell type annotation· 3 Research Focus:differential expression· 3 Organism:human· 3 Assay:bulk RNA-seq· 2 Research Focus:development & morphogenesis· 2 Research Focus:host-pathogen & immune response· 2

Frequent Co-Authors

J. A. Kropski· 5 shared J. J. Gokey· 3 shared T. Sherrill· 3 shared W. Han· 3 shared A. S. McCall· 2 shared B. W. Richmond· 2 shared C. L. Calvi· 2 shared D. Nichols· 2 shared

Activity by Year

2021-2023 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 40 mutual co-author links

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5 shared papersCell Biology

J. A. Kropski

Mutual co-authors in cluster

Author profile →

6 papers

Recent·Most cited

Hypoxia-inducible factor-2 (HIF2) regulates alveolar regeneration after repetitive injury

A. S. McCall, H. Tanjore, A. Burman, T. Sherrill, M. Chapman, C. L. Calvi, J. Camarata, R. P. Hunt, D. Nichols, N. Banovich, W. E. Lawson, J. J. Gokey, J. A. Kropski, T. S. Blackwell

Inhibiting hypoxia-inducible-factor 2 (HIF2) promotes functional lung alveolar epithelial repair following recurrent injury.

bioRxiv · Sep 17, 2023 · PreprintCell Biology

Epithelial Yap/Taz are required for functional alveolar regeneration following acute lung injury

G. T. DiGiovanni, W. Han, T. Sherrill, C. J. Taylor, D. S. Nichols, N. M. Geis, U. K. Singha, C. L. Calvi, A. S. McCall, M. M. Dixon, Y. Lui, J.-H. Jang, S. S. Gutor, V. V. Polosukhin, T. S. Blackwell, , J. J. Gokey

This study shows that Yap/Taz in AT2 cells are essential for alveolar regeneration and preventing fibrosis after lung injury.

bioRxiv · Jun 22, 2023Cell Biology

Repetitive Sulfur Dioxide Exposure in Mice Models Post-Deployment Respiratory Syndrome

S. S. Gutor, R. I. Salinas, D. S. Nichols, J. M. R. Bazzano, W. Han, J. J. Gokey, G. Vasiukov, J. D. West, D. C. Newcomb, A. E. Dikalova, B. W. Richmond, S. I. Dikalov, T. S. Blackwell, V. V. Polosukhin

This study develops a mouse model of repetitive sulfur dioxide exposure that mimics post-deployment respiratory syndrome features, identifying oxidative stress as a mediator of pulmonary vascular disease.

bioRxiv · May 15, 2023Pathology

Alveolar repair following lipopolysaccharide-induced injury requires cell-extracellular matrix interactions

J. M. S. Sucre, F. Bock, N. M. Negretti, J. T. Benjamin, P. M. Gulleman, X. Dong, K. T. Ferguson, C. S. Jetter, W. Han, Y. Liu, S. Kook, J. Gokey, S. H. Guttentag, J. A. Kropski, T. S. Blackwell, , E. J. Plosa

This study shows that β1-containing integrins are essential for alveolar repair after injury, as their absence leads to emphysema and impaired differentiation of AT2 cells into mature type 1 alveolar epithelial cells.

bioRxiv · Aug 5, 2022 · PreprintCell Biology

A Unique Cellular Organization of Human Distal Airways and Its Disarray in Chronic Obstructive Pulmonary Disease

S. Rustam, Y. Hu, S. B. Mahjour, S. H. Randell, A. F. Rendeiro, H. Ravichandran, A. Urso, F. d'Ovidio, F. J. Martinez, B. Richmond, V. Polosukhin, J. A. Kropski, T. S. Blackwell, O. Elemento, R. Shaykhiev

This study identifies a unique epithelial cell population in human distal airways and shows its reduction in COPD, linked to IFN-gamma signaling.

bioRxiv · Mar 16, 2022Cell Biology

Secretory cells are the primary source of pIgR in small airways

J. B. Blackburn, J. A. Schaff, S. Gutor, R.-H. Du, D. Nichols, T. Sherrill, A. J. Gutierrez, M. K. Xin, N. Wickersham, Y. Zhang, M. J. Holtzman, L. B. Ware, N. E. Banovich, J. A. Kropski, T. S. Blackwell, B. W. Richmond

The paper identifies secretory cells as the main source of pIgR in airways and shows reduced pIgR protein expression, not cell numbers, contributes to SIgA loss in COPD.

bioRxiv · Nov 10, 2021Cell Biology