Author

W. C. K. Wang

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:development & morphogenesis· 2 Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Assay:imaging· 1

Frequent Co-Authors

D. F. Schaeffer· 2 shared S. Patil· 2 shared A. M. Y. Zhang· 1 shared C. H. Borchers· 1 shared E. Rideout· 1 shared G. H. Su· 1 shared H. H. Cen· 1 shared I. S. Wood· 1 shared

Activity by Year

2022-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersCancer Biology

D. F. Schaeffer

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2 papers

Recent·Most cited

Acvr1b loss increases formation of pancreatic precancerous lesions from acinar and ductal cells of origin

K. Saeki, I. S. Wood, W. C. K. Wang, S. Patil, Y. Sun, D. F. Schaeffer, G. H. Su, J. L. Kopp

This study shows that Acvr1b loss in combination with Kras mutation promotes precancerous lesions from both acinar and ductal cells in the pancreas, but the IPMN-like phenotype differs when mutations occur in all cells.

bioRxiv · Jun 8, 2023 · PreprintCancer Biology

Hyperinsulinemia acts via acinar insulin receptors to initiate pancreatic cancer by increasing digestive enzyme production and inflammation

A. M. Y. Zhang, Y. H. Xia, J. S. H. Lin, K. H. Chu, W. C. K. Wang, T. J. J. Ruiter, J. C. C. Yang, N. Chen, J. Chhuor, S. Patil, H. H. Cen, E. Rideout, V. R. Richard, D. F. Schaeffer, R. P. Zahedi, , J. Kopp

bioRxiv · May 6, 2022Cancer Biology