W. M. C. Awata

This study shows that CCL5/CCR5 contributes to aldosterone-induced hypertension, vascular injury, and renal damage by promoting inflammation and oxidative stress.

This study shows ethanol-induced vascular dysfunction is mediated by AT1 receptor activation and mitochondrial dysfunction, which can be mitigated by losartan.

This study shows that progranulin regulates blood pressure and vascular tone through EphrinA2, Sortilin1 receptors, and eNOS activation, with PGRN deficiency causing hypertension and vascular dysfunction.

This study examines vascular function and blood pressure in a mouse model of Kawasaki disease, finding vascular dysfunction linked to COX-derived products, oxidants, and NO bioavailability.