Author

Y. Onodera

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:chromatin & epigenetic regulation· 2 Approach:experimental assay· 2 Organism:human· 2 Research Focus:cancer & tumor microenvironment· 1 Assay:ChIP-seq· 1 Assay:DNA methylation· 1 Assay:proteomics· 1

Frequent Co-Authors

A. Hashimoto· 1 shared A. Sakurada· 1 shared D. Matsumaru· 1 shared F. Katsuoka· 1 shared H. Anzawa· 1 shared H. Handa· 1 shared H. Kitamura· 1 shared H. Motohashi· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperCell Biology

A. Hashimoto

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2 papers

Recent·Most cited

p53 secures the normal behavior of H3.1 histone in the nucleus by regulating nuclear phosphatidic acid and EZH2 during the G1/S phase

T. Oikawa, J. Hasegawa, H. Handa, N. Ohnishi, Y. Onodera, A. Hashimoto, J. Sasaki, T. Sasaki, K. Ueda, H. Sabe

This study shows p53 regulates H3.1 histone behavior during G1/S phase by modulating nuclear phosphatidic acid and EZH2 levels, preventing H3.1 from being trimethylated at the nuclear envelope.

bioRxiv · Jun 27, 2023 · PreprintCell Biology

Enhancer Remodeling Promotes Tumor-Initiating Activity in NRF2-Activated Non-Small Cell Lung Cancers

K. Okazaki, H. Anzawa, Z. Liu, N. Ota, H. Kitamura, Y. Onodera, M. M. Alam, D. Matsumaru, T. Suzuki, F. Katsuoka, S. Tadaka, I. Motoike, M. Watanabe, A. Sakurada, Y. Okada, , H. Motohashi

The paper identifies NRF2-CEBPB cooperativity in promoting tumor-initiating activity in NRF2-activated NSCLCs through enhancer remodeling and the NRF2-NOTCH3 regulatory axis.

bioRxiv · Apr 24, 2020Cancer Biology