Author

Y. V. Kwon

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Organism:Drosophila· 3 Approach:experimental assay· 3 Research Focus:cell type annotation· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:development & morphogenesis· 1 Organism:human· 1 Research Focus:protein folding & dynamics· 1

Frequent Co-Authors

A. J. H. Cabrera· 1 shared B. M. Gumbiner· 1 shared H. Chae· 1 shared H.-J. Choi· 1 shared I. K. Pranoto· 1 shared J. Lee· 1 shared J.-E. Kim· 1 shared J.-W. Nam· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 17 mutual co-author links

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1 shared paperCell Biology

A. J. H. Cabrera

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Comparative Interactome Analysis of α-arrestin Families in Human and Drosophila

K.-T. Lee, I. K. Pranoto, S.-Y. Kim, H.-J. Choi, N. B. To, H. Chae, J.-Y. Lee, J.-E. Kim, Y. V. Kwon, J.-W. Nam

This study constructs and compares interactomes of α-arrestins in human and Drosophila, identifying conserved and species-specific binding partners and revealing TXNIP's role in modulating chromatin structure.

bioRxiv · Jun 11, 2023 · PreprintCell Biology

The roles of distinct Ca2+ signaling mediated by Piezo and inositol triphosphate receptor (IP3R) in the remodeling of E-cadherin during cell dissemination

A. J. H. Cabrera, B. M. Gumbiner, Y. V. Kwon

This study shows that E-cadherin remodeling during cell dissemination in Drosophila is controlled by two distinct Ca2+ signaling pathways mediated by IP3R and Piezo.

bioRxiv · Nov 10, 2021 · PreprintCell Biology

Tumors Negate the Action of ImpL2 by Elevating Wingless

J. Lee, K. G.- L. Ng, K. M. Dombek, Y. V. Kwon

The paper shows tumors counteract the wasting factor ImpL2 by increasing Wingless, which supports tumor growth and suggests targeting the Wnt/Wg pathway could treat cancers with elevated IGFBPs.

bioRxiv · Sep 16, 2020Cancer Biology