Author

Y.-E. Lin

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 1 Cell Biology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:cell signaling & trafficking· 2 Research Focus:cell type annotation· 2 Assay:proteomics· 2 Research Focus:differential expression· 1 Research Focus:host-pathogen & immune response· 1 Research Focus:metabolism & bioenergetics· 1

Frequent Co-Authors

E. Jaimon· 2 shared F. Tonelli· 2 shared S. R. Pfeffer· 2 shared C. Themistokleous· 1 shared C.-C. Chen· 1 shared C.-C. Hsu· 1 shared C.-T. Ho· 1 shared D. R. Alessi· 1 shared

Activity by Year

2021-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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2 shared papersCell Biology

E. Jaimon

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Pathogenic LRRK2 mutations cause loss of primary cilia and Neurturin in striatal Parvalbumin interneurons

Y.-E. Lin, E. Jaimon, F. Tonelli, S. R. Pfeffer

The paper shows that LRRK2 mutations cause loss of primary cilia and Neurturin in striatal Parvalbumin interneurons, impairing neuroprotective signaling.

bioRxiv · Jun 17, 2024Cell Biology

Endogenous LRRK2 and PINK1 function in a convergent neuroprotective ciliogenesis pathway in the brain

E. Bagnoli, Y.-E. Lin, S. Burel, E. Jaimon, O. Antico, C. Themistokleous, J. Nikoloff, I. Morella, J. Watzlawik, F. Fiesel, W. Springer, F. Tonelli, S. Brooks, S. Dunnett, R. Brambilla, , M. Muqit

This study shows LRRK2 and PINK1 function in parallel neuroprotective pathways affecting ciliogenesis in Parkinson's disease, suggesting shared therapeutic targets.

bioRxiv · Jun 11, 2024Biochemistry

Dietary Exposure to Antibiotic Residues Facilitates Metabolic Disorder by Altering the Gut Microbiota and Bile Acid Composition

R.-A. Chen, W.-K. Wu, S. Panyod, P.-Y. Liu, H.-L. Chuang, Y.-H. Chen, Q. Lyu, H.-C. Hsu, T.-L. Lin, T.-C. D. Shen, Y.-T. Yang, H.-B. Zou, H.-S. Huang, Y.-E. Lin, C.-C. Chen, , L.-Y. Sheen

This study shows that low-dose antibiotic residue exposure in early life alters gut microbiota and bile acid composition, leading to long-term metabolic disorders in mice.

bioRxiv · Jul 6, 2021Microbiology