Author

Yasmin Razia

Washington University in St. Louis

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

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Neuroscience· 1

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Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1 Approach:statistical model· 1

Frequent Co-Authors

Faiza BatooliD· 1 shared Gareth ChapmaniD· 1 shared Haley JetteriD· 1 shared Harrison W. GabeliD· 1 shared James E. HuettneriD· 1 shared John R. EdwardsiD· 1 shared Julianna J. DetermaniD· 1 shared Kristen L. KrolliD· 1 shared

Activity by Year

2025-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperNeuroscience

Faiza BatoolORCID

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1 paper

Recent·Most cited

Tatton-Brown-Rahman-Syndrome-associated DNMT3A mutations de-repress cortical interneuron differentiation to disrupt neuronal network function

Gareth Chapman, Julianna J. Determan, John R. Edwards, Faiza Batool, James E. Huettner, Ramachandran Prakasam, Sydney R. Crump, Yasmin Razia, Sofia Malik, Travis E. Law, Haley Jetter, Harrison W. Gabel, Kristen L. Kroll

This study shows that DNMT3A mutations in TBRS disrupt cortical interneuron differentiation, leading to neuronal network dysfunction and contributing to intellectual disability and autism.

bioRxiv · Jun 24, 2025 · PreprintNeuroscience