Author

Faiza Batool

Washington University in St. LouisORCID 0000-0002-9045-1774Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cell Biology· 1 Developmental Biology· 1 Neuroscience· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 3 Research Focus:differential expression· 3 Organism:human· 3 Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Research Focus:gene regulatory network inference· 2 Approach:statistical model· 2 Research Focus:cell-cell communication· 1

Frequent Co-Authors

Gareth ChapmaniD· 2 shared Haley JetteriD· 2 shared Julianna J. DetermaniD· 2 shared Kristen L. KrolliD· 2 shared Sofia MalikiD· 2 shared Sydney R. CrumpiD· 2 shared Caleb Valentine· 1 shared Harrison W. GabeliD· 1 shared

Activity by Year

2025-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 42 mutual co-author links

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2 shared papersDevelopmental Biology

Gareth ChapmanORCID

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Author profile →

3 papers

Recent·Most cited

Intellectual disability risk gene RFX4 regulates cortical neurogenesis by restraining neuronal differentiation

Julianna J. Determan, Gareth Chapman, Sydney R. Crump, Faiza Batool, Sofia Malik, Taranjit S. Gujral, William Buchser, Caleb Valentine, Serena N Elia, Monica F. Sentmanat, Xiaoxia Cui, Haley Jetter, Kristen L. Kroll

This study identifies RFX4's role in cortical development by showing it restrains neurogenesis and regulates synaptic gene expression, with distinct effects from haploinsufficiency.

bioRxiv · Feb 17, 2026 · PreprintDevelopmental Biology

mTORC2-mediated cell-cell interactions promote BMP4-induced WNT activation and mesoderm differentiation

Li Tong, Faiza Batool, Yueh-Ho Chiu, Priscilla Di Wu, Yudong Zhou, Xiaolun Ma, Santosh S. Atanur, Wei Cui

This study shows mTORC2 is required for BMP4-induced WNT activation and mesoderm differentiation through cell-cell interactions in hESCs.

Stem Cell Reports · Oct 16, 2025Peer-reviewedCell Biology

Tatton-Brown-Rahman-Syndrome-associated DNMT3A mutations de-repress cortical interneuron differentiation to disrupt neuronal network function

Gareth Chapman, Julianna J. Determan, John R. Edwards, Faiza Batool, James E. Huettner, Ramachandran Prakasam, Sydney R. Crump, Yasmin Razia, Sofia Malik, Travis E. Law, Haley Jetter, Harrison W. Gabel, Kristen L. Kroll

This study shows that DNMT3A mutations in TBRS disrupt cortical interneuron differentiation, leading to neuronal network dysfunction and contributing to intellectual disability and autism.

bioRxiv · Jun 24, 2025 · PreprintNeuroscience