Author

Zinnia Saha

Stanford Medicine

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1

Frequent Co-Authors

Julia A. KaltschmidtiD· 2 shared Amr A. MahrousiD· 1 shared Atharv PanditraoiD· 1 shared Charles J. HeckmaniD· 1 shared Cheyanne M. LewisiD· 1 shared David J. BennettiD· 1 shared Eric Tianjiao ZhaoiD· 1 shared Irene Lorenzo LlorenteiD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersNeuroscience

Julia A. KaltschmidtORCID

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2 papers

Recent·Most cited

Loss of Sall3 eliminates presynaptic inhibition of sensory motor circuits and impairs adaptive motor behavior

Jennifer L. Shadrach, Amr A. Mahrous, Róbert Pálovics, Zinnia Saha, Richard H. Roth, Atharv Panditrao, Vanessa W. Y. Kan, Mark A. Gradwell, Victoria E. Abraira, Irene Lorenzo Llorente, Jun Ding, Tony Wyss‐Coray, David J. Bennett, Charles J. Heckman, Julia A. Kaltschmidt

This study shows that Sall3 is essential for forming GABApre synapses on Ia afferents, and its loss disrupts motor behavior by impairing presynaptic inhibition.

bioRxiv · Aug 31, 2026 · PreprintNeuroscience

Enteric glutamatergic interneurons regulate intestinal motility

Ryan Hamnett, Jacqueline L. Bendrick, Zinnia Saha, Keiramarie Robertson, Cheyanne M. Lewis, Jack H. Marciano, Eric Tianjiao Zhao, Julia A. Kaltschmidt

This study identifies glutamatergic neurons as key regulators of intestinal motility by showing their synaptic connections and functional roles in gut transit.

Neuron · Feb 20, 2025Peer-reviewedNeuroscience