Author

Debora Comai

University of TurinORCID 0000-0001-5201-5915Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:clustering· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Assay:electronic health records· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

Alessandra RaspantiiD· 2 shared Antonia GurgoneiD· 2 shared Giulia SagonaiD· 2 shared Maurizio GiustettoiD· 2 shared Riccardo PizzoiD· 2 shared Tommaso PizzorussoiD· 2 shared Alessio NocentiniiD· 1 shared Andrea Macioce· 1 shared

Activity by Year

2022-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 41 mutual co-author links

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2 shared papersNeuroscience

Alessandra RaspantiORCID

Mutual co-authors in cluster

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2 papers

Recent·Most cited

NeuroscienceProgress in NeurobiologyPeer-reviewedAug 19, 2026

Postnatal Sensory Experience and Barrel Cortex Alterations Anticipate Autistic Traits in a Mouse Model of Cdkl5 Deficiency Disorder

Alessandra Raspanti, Riccardo Pizzo, Antonia Gurgone…·Maurizio Giustetto

This study shows that CDKL5 deficiency in mice leads to sensory processing defects in the barrel cortex and autistic traits, which can be rescued by neonatal CDKL5 replacement.

1 citation

mouseelectronic health records

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NeuroscienceNeuropsychopharmacologyPeer-reviewedAug 9, 2022

mGluR5 PAMs rescue cortical and behavioural defects in a mouse model of CDKL5 deficiency disorder.

Antonia Gurgone, Riccardo Pizzo, Alessandra Raspanti…·Maurizio Giustetto

This study shows that mGluR5 positive-allosteric modulators correct synaptic and behavioural defects in a CDKL5 deficiency disorder mouse model and human patients.

16 citations

mouseexperimental assay

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